Managing Infections with HBOT
HBOT acts as a bactericidal agent by impairing bacterial metabolism. It also improves phagocytosis by white blood cells. Supplying oxygen to the immune mechanism of the body improves the function and efficiency of the immune process. HBOT produces free radicles that are toxic to micro-organisms. Adequate tissue oxygenation provided by HBOT improves the action of antibiotics. HBOT also inhibits exotoxin production as seen in clostridium perfringens.
Managementanagement for Infections
HBOT is effective against common necrotizing infections, such as:
- Clostridial and non-clostridial cellulitis
- Progressive bacterial gangrene, usually seen with staphyloccocal and streptococcal infection usually associated with ulcers
- Necrotizing fasciitis due to streptoccocal infection
- Myonecrosis (non-clostridial)
- Gas gangrene usually associated with trauma or surgery, but may occur spontaneously due to a variety of anaerobic organisms, and is also referred to as clostridial myonecrosis (gram positive)
Other Infections Aided by HBOT:
- Candida albicans
- Phycomycotic fungal infections which may give rise to necrosis of the fascial layers overlying muscles (necrotizing fasciitis)
- Nosocomial infections, i.e. pseudomonas
HBOT and AIDS:
HBOT produces reactive oxygen intermediates, which are virucidal against enveloped viruses. Free radicles produced by HBOT can penetrate the lipid covering of viruses (toxic effect of these free radicals can be countered in normal cells with magnesium).
HBOT also enhances the immune system. Lastly, HBOT is effective against the opportunistic infections in HIV.
- George Allibone M.D.